We have speculated in the past that the A− trait may, nevertheless, be somehow expressed in N and NP carriers of the trait, i.e., in NPA types who lack the trait of aggression. The trait could be expressed in some subtle behavioral condition (for example, susceptibility to panic attacks in N types), but it could also be expressed in a physical condition, like eczema.
We speculated that in a sanguine habitancy with high A trait (say, France), individuals with eczema would be mostly ones with A− trait expressed (high S score on the NPA test). In a sanguine habitancy with lower A trait (say, Germany), there would be many N and NP types with eczema -- carriers of A− trait -- who would have lower S scores on the NPA test.
We only started collecting NPA Test data for eczema last year, so we have only a small number of results, but so far 5 "French" subjects have a very high mean S score of 67, compared to 3 "English" subjects with a low mean S score of 20.
Finally, we address the well-known association of schizophrenia (SCZ) with eczema and our prior speculations that in some families SCZ is related to the A− trait. In a particular family where there are individuals of various NPA types and individuals with SCZ and eczema, N and NP individuals with eczema could identified as carriers of the A− trait, as well as carriers of susceptibility to SCZ, even though they may not have SCZ or A− trait expressed themselves.
These speculations are far removed from the conventional wisdom of the research community. The concept that humans have a single gene that can completely inhibit the trait of aggression (allowing the existence of N and NP types) does not seem to have occurred to anyone.
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