Here the study is in Iran it sounds like. The habitancy there we think is rather demonstrative, possibly polymorphic, possibly corybantic with a strong feeling based on national behavior that they are corybantic.
So what can this mean about their study which found that autistics who carried DRD3 gly allele were more responsive to a dopamine blocker. (First this is found so often that it is now a standard of care in use of this drug and others like them.)
Why would this be and what does it say about the cause of autism? If those who had blocked receptors already had less effect from a blocking drug, that makes sense. But what causes autism? If some individuals had blocked receptor and some didn't, perhaps the receptor didn't cause autism since even those who already had blocked receptors still had autism.
Or maybe entanglement is at work. One can have autism if he has blocked DRD3 receptors (and thus A trait) only if he also has unblocked DRD2 or perhaps DRD4 or other dopamine receptors (and thus N trait) or perhaps other genes (like COMT) that modify A trait (thus A- trait).
So the population of autistic individuals in this study are entangled because they are heterogeneous for the dopamine receptors of various types such that studying only one of the receptors DRD3 in this case is not sufficient to characterize the population. In NPA terms, some of the individuals who are autistic are N types and some are A types.
And unblocked dopamine receptors still are major candidates for autism causes.
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